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Additionally, defects in mitophagy and mitochondrial DNA mutations further exacerbate disease progression by disrupting cellular quality control and energy metabolism (Fig

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On the translocation of bacteria and their lipopolysaccharides between blood and peripheral locations in chronic, inflammatory diseases: the central roles of LPS and LPS-induced cell death

Schaffer S, Asseburg H, Kuntz S, Muller WE, Eckert GP (2012) Effects of polyphenols on brain ageing and Alzheimer's disease: focus on mitochondria

Favorable activity and safety profile of memory-enriched CD19-targeted chimeric antigen receptor T-cell therapy in adults with high-risk relapsed/refractory ALL