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Apoptosis is a caspase-dependent process activated by intrinsic (e.g., mitochondrial outer membrane permeabilization) or extrinsic (e.g., death receptor activation) stimuli

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10.1097/OPX.0000000000001826 25 ChakrabortyU.ChandraA

However, when activated by various pathological stimuli, including neurodegeneration, microglia can adopt a proinflammatory phenotype, leading to the release of cytokines, chemokines, and other mediators that perpetuate inflammation and contribute to neuronal loss [37,38]

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