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Mitochondrial injury directly impairs the function of alveolar epithelial cells, thereby triggering local inflammatory responses and promoting fibroblast activation driven by TGF-, a process that plays a critical role in the development of pulmonary fibrosis (Liu and Chen, 2017

The finding that levels of free glutamine and glutamate in human milk are relatively high, display unique dynamics along lactation, and are associated with infant anthropometrics urges the need to understand the functions that these FAAs could have during infant development

Altering ethanol pharmacokinetics to treat alcohol use disorder: can you teach an old dog new tricks

3 Mechanisms underlying cell death in cardiovascular disease Cell death is a central pathological event in CVDs, contributing to the progression of conditions such as atherosclerosis, myocardial infarction, and heart failure (Figure 2) ( Figure 2 3.1 Programmed cell death in cardiovascular diseases PCD refers to tightly regulated cellular processes involving molecular pathways that orchestrate cell demise ( Apoptosis, a caspase-mediated process, is pivotal in both acute and chronic cardiovascular injury ( Autophagy is primarily a survival mechanism, but excessive autophagy contributes to cell death under stress conditions, such as hypoxia or oxidative stress ( Iron-dependent lipid peroxidation characterizes ferroptosis, which is distinct from other PCD modalities (102)

PMID: 30342045

AKI has been associated with acute changes in kidney function and long-term prognosis, including progression to CKD, cardiovascular disease, persistent function, and even death (147, 148)