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Under the combined influence of inflammatory mediators and oxidative stress, chondrocytes undergo chondroaging, characterized by mitochondrial dysfunction, disordered cellular metabolism, reduced SIRT1 activity, and impaired autophagy

It recruits IRS-1 and PI3K, leading to the phosphorylation of mTOR, which drives ribosomal activity and muscle hypertrophy

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Without adequate antioxidant protection, these radicals can damage proteins, DNA, and cellular membranes

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This fluidity is biologically important, influencing membrane transport