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Stem Cell Research & Therapy , 12 (1)

Recent elegant work has definitively identified that TP53 missense mutations in myeloid malignancies result in a dominant-negative effect without evidence of neomorphic gain-of-function activities, ultimately leading to a selection advantage when exposed to DNA damage.12 Thus, restoring wild-type function in TP53 mutant clones would be of profound beneficial impact

one is found in the bacterial cytoplasm (internal urease), and the other is present on the bacterial surface (external urease) (Sharndama and Mba 2022)

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In another aspect of this embodiment, the disorder is an excitotoxic disease involving oxidative cell death