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In patients with RA remission, the level of phosphatidyl acid (PA(28:0)) in synovial fluid significantly increased, which stabilized SCAP-SREBP complex on the endoplasmic reticulum by binding to Insig protein, and inhibited the expression of HMGCR, a key enzyme in cholesterol synthesis (76, 78, 79)

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Presynaptic cytoplasmic Ca 2+ can be depleted by several complementary mechanisms, including sarcoendoplasmic reticulum Ca 2+ -ATPase (SERCA) to the endoplasmic reticulum (ER), plasma membrane Ca 2+ -ATPase to the extracellular space, and MCU to the mitochondrial matrix [51,52,53]