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and USP18 leads to sorafenib resistance by de-ISGylating NCOA4 and mediating its degradation, which inhibits ferritinophagy and ferroptosis [111]

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Its integrity not only determines cellular tolerance to oxidative damage but is also closely related to metabolic adaptability, drug sensitivity, and the aging process

CD38 and MGluR1 as possible signaling molecules involved in epileptogenesis: a potential role for NAD(+) homeostasis

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