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Current understanding attributes ferroptosis in ischemic stroke to several factors, including intracellular iron accumulation, lipoxygenase activation, GSH depletion, GPX4 inactivation, and system Xc disruption (130) ( Figure 3 )

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3,3 -diindolylmethane promotes bdnf and antioxidant enzyme formation via trkb/akt pathway activation for neuroprotection against oxidative stress-induced apoptosis in hippocampal neuronal cells

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PMID: 41548684 J Inflamm Res Decrease of NAD+ Inhibits the Apoptosis of OLP T Cells via Inducing Mitochondrial Fission