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Similar to the early production of other viral vectors [26], a scale-out approach was employed with dozens of 10-layer cell stacks and several rounds of ultracentrifugation over multiple days

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& Dimery, I

It is also worth noting that MK-677 and ipamorelin target the same receptor GHS-R1a meaning they are pharmacological competitors rather than mechanistically complementary agents

10.4049/jimmunol.2000500 Summary Keywords ferroptosis, cancer, pharmacology, post-translational modifications, p53, cancer therapy Citation Zhang L, Hou N, Chen B, Kan C, Han F, Zhang J and Sun X (2022) Post-Translational Modifications of p53 in Ferroptosis: Novel Pharmacological Targets for Cancer Therapy