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4 Core pathophysiological mechanisms of DPN DPN results from the convergence of metabolic, inflammatory, vascular, and neuronal disturbances driven by chronic hyperglycemia

In RA patients, CD56 + NKp44 + NK cells secret high level of IL-22 to promote the proliferation of fibroblast-like synoviocytes and lead to synovial hyperplasia, resulting in cartilage and bone destruction (76)