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Importantly, the OKT10 mAb targets a different epitope to daratumumab, which may allow it to be effective in patients receiving daratumumab, potentially mitigating resistance [134]

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The splice variants IGF-1Eb and IGF-1Ec encode the alternative proIGF-1Eb and proIGF-1Ec forms, which were insensitive to modulation of glycosylation

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