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Thus, there is no reason to discard a major role of heroin per se in the early response to heroin in humans, contrary to the widely held notion of heroin as a mere pro-drug

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The question arises, can anything be done to limit this provision and/or prevent its catabolism in a way that would reduce the amount of the intended products, i.e., yielding NADPH, nucleotides, folate, hexosamines, serine, glycine, TGs, PLs, lactate, in a bid to limit cancers potential

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