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In conclusion, lipid accumulation plays multiple roles in the pathogenesis of renal tubular injury of DKD, including impairing tubular cell function, increasing oxidative stress and mitochondrial dysfunction, inducing inflammation, promoting tubular cell death and exacerbating hypoxia

Suppression of enteroendocrine cell glucagon-like peptide (GLP)-1 release by fat-induced small intestinal ketogenesis: a mechanism targeted by Roux-en-Y gastric bypass surgery but not by preoperative very-low-calorie diet

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You might have heard conflicting information about whether certain antioxidants play a role in this process

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