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Early studies on Gly-His-Lys (GHK), first discovered in human plasma in the 1970s, demonstrated their ability to modulate gene expression, stimulate collagen synthesis, and accelerate wound healing, sparking broad interest in these bioactive molecules [23]

Neither livers nor embryos lacking TrxR1 exhibit compensatory induction of Gsr

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Several researchers [539] provide further reflection on the autism-epilepsy phenotype and a growing body of evidence for shared neuronal networks that can account for both ASD and epilepsy

have been shown to exhibit relatively high activity in methylglyoxal formation and its excretion during bacterial growth, thereby potentially influencing the intestinal balance of glycation products in the host (123127)

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