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10.1186/s12964-021-00738-7 Summary Keywords apoptosis, Bcl-2, hydroxysafflor yellow A, sepsis-induced intestinal barrier dysfunction, SOD2 Citation Fei J, Xu C, Chen C, Chen Q, Wu Z, Wang Y, Bao D and Shao S (2026) Hydroxysafflor yellow A attenuates sepsis-induced intestinal barrier dysfunction by modulating Bcl-2/SOD2-mediated mitochondrial apoptosis

A plethora of PASC clinical symptoms and related metabolic impairments indicate involvement of different pathobiological mechanisms such as (i) virus-induced hypoxia/Warburg effect, (ii) iron (Fe)-redox dysregulation (FeRD), (iii) m- Dys and altered bioenergetics, (iv) oxidative stress (OxS) and cellular damage, (v) immuno-pathogenesis and hyperinflammation, (vi) autoimmunity, (vii) dysbiosis, (viii) re-activation of latent pathogens, (ix) persistent viral reservoirs, and (x) viral-hijacked host cellular factors 22,27,137,138

In particular, populations of somatostatin neurons occur in the arcuate nucleus, the hippocampus, and the brainstem nucleus of the solitary tract

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Differential abundance and genus analyses (percent read abundance) were performed