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Protective isolation may be required in aplastic anemia when immune responses are most compromised

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The pathogenesis of AD is multifactorial, involving the accumulation of amyloidbeta (A) plaques, neurofibrillary tangles (NFTs) composed of hyperphosphorylated tau protein, oxidative stress, and chronic neuroinflammation (GarcaMorales et al

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AOD 9604, because it works specifically on adipose tissue through lipolytic pathways, theoretically targets fat preferentially