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In this regard, Eren et al

Research has identified some genetic mechanisms that might result in the development of drug resistance against targeted therapies, which may include secondary mutations, either upstream or downstream of effector activation, and could result in the bypass of certain biological pathways, in addition to epigenetic changes ( Figure 4 )

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Key genes and signaling pathways, such as the Wnt/-catenin, MET, and signal transducer and activator of transcription 3 (STAT3) pathways, have been identified

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