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( N - tert -butoxycarbonyl)-L-selenocysteine methyl ester was prepared in situ 86 in methanol or phosphate buffer

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AST was measured based on the methods of Henry et al [37]

System xc controls cystine uptake and its subsequent reduction to cysteine, the shared substrate for both GSH synthesis and H 2 S biosynthesis, creating a metabolic branch point where RSS production and antioxidant defense compete for limited cysteine pools

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Patients were treated with 16 weeks and magnetic resonance imaging (MRI)-proton density fat fraction [MRI-PDFF] and homeostasis model assessment of insulin resistance [HOMA-IR]) and homeostasis model assessment of insulin resistance (HOMA-IR) fibro-inflammation markers (alanine aminotransferase [ALT], corrected T1 [cT1], keratin-18 [K-18] M65, and N-terminal type III collagen pro-peptide [Pro-C3]) was applied