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Once activated, ERK1/2 is transferred to the nucleus, where it phosphorylates transcription factors such as c-Myc and c-Fos, leading to the expression of genes responsible for cellular regulations such as cell proliferation, differentiation, and migration

By disrupting FOXO4-p53 interactions, it promotes apoptosis in senescent cells, reducing inflammation and improving cellular health (3)

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Research has examined its influence on HGF-dependent signaling processes, receptor-mediated signal transduction, and downstream regulatory pathways associated with c-Met activation

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