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Altogether, these data support the cell-autonomous nature of thyroid dyshormonogenesis phenotype observed in nrf2a5 homozygous mutant zebrafish embryos

if caspase-8 activity is inhibited, active RIPK1 forms a death-inducing signaling complex (the necrosome), which also includes RIPK3 and mixed lineage kinase domain-like protein (MLKL) [53]

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Serological profiling of the EBV immune response in chronic fatigue syndrome using a peptide microarray