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It is a direct blockade of one of the primary engines driving chronic inflammatory states

Antioxidant capacity in the lung is substantially reduced and oxidative stress can persist long after the cessation of cigarette smoking or an acute exacerbation of COPD owing to continued production of ROS from endogenous sources [9, 12]

were the first to systematically demonstrate that cuproptosis is a copper-dependent cell death mode induced by mitochondrial respiration and proteotoxic stress, highlighting the crucial role of copper homeostasis in cancer and other diseases (Tsvetkov et al., 2022)

Early exposure to general anesthesia disturbs mitochondrial fission and fusion in the developing rat brain

Clin Transl Med 13(1):e1150

Segovia G, Porras A, Del Arco A, Mora F