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Systems Biology in Cancer Research and Drug Discovery

Biochemical reports also demonstrated that enhanced oxidative stress, dysregulated apoptosis, and extensive inflammatory reactions mediated by overexpressed inflammatory factors [such as tumor necrosis factor (TNF-), interleukin 1 (IL-1), and interleukin 6 (IL-6)] were served as the underlying mechanisms for the occurrence of various CSD-relevant diseases (810)

Li Y, Huang R, Li X, Li X, Yu D, Zhang M

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PMID: 41203627 Acta Pharmacol Sin Targeting PPAR activation sensitizes glioblastoma cells to temozolomide and reverses acquired resistance by inhibiting H3K18 lactylation

Similarly, targeting ERK, the final downstream kinase of the MAPK signaling pathway, has shown potential