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Cognitive impairment in Gdi1-deficient mice is associated with altered synaptic vesicle pools and short-term synaptic plasticity, and can be corrected by appropriate learning training

Cellular mechanisms and regulation of quiescence

Treatment failure (OR 1.8, 95%CI:1.3-4.9, p =0.002) and presence of HE (OR4.4, 95%CI:1.3-5.6, p =0.044) were associated with progression of PHT

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Rungelrath, V., Kobayashi, S