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Mapping and refactoring pathway control through metabolic and protein engineering: the hexosamine biosynthesis pathway

Highdose folic acid improves endothelial function by increasing tetrahydrobiopterin and decreasing homocysteine levels

Its mechanism of action upstream stimulation of pituitary GH secretion rather than exogenous GH replacement preserves the regulatory architecture of the hypothalamic-pituitary axis, making it a pharmacologically distinct tool from direct rhGH administration in ageing research models