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(2024) 10:e39721

erecta and T

Mechanistically, these effects appeared to be mediated through mitochondrial uncoupling, AMPK activation, modulation of mTOR and PPAR signaling, improved mitochondrial bioenergetics, antioxidant defense and global metabolic reprogramming (Fig

Forero-Quintero, L

105 DNA repair HK2 exhibits moonlighting functions in the nucleus, with its overexpression increasing chromatin accessibility at DNA repair sites, thereby reducing DNA double-strand breaks