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In addition to downregulation of injury and inflammation-related pathways, GSEA also revealed suppression of adipogenesis-related pathways in the liver of GPR146-deficient mice, suggesting that GPR146 may promote hepatic steatosis via PPAR-mediated transcriptional programs (Fig

As the substrate and, thereby, the fiber content of our interventions was similar, the pasteurized sauerkraut microbes themselves or their products might be responsible for our findings

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Concomitant decreases of ascorbate (AA) and reduced glutathione (GSH) were also detected in both BALF and lung tissues

Sies H (1985) Oxidative stress: introductory remarks