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The dysregulation of glycolysis in these cells has been identified as a significant initiator of neuronal impairments in AD [66]

Dual Inhibition of CDK4/6 and XPO1 induces senescence with acquired vulnerability to CRBN-based PROTAC drugs

Studies have also demonstrated that mice expressing ApoE4 in tauopathy models exhibit more severe tau-induced neurodegeneration compared to other isoforms, while mice lacking ApoE are protected from this effect (Shi et al., 2017)

S2A), while some others showed exon duplication such as CaGSTU 14 and CaGSTU 47 (Fig

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