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Therefore, aberrant activation of the PI3K/AKT/mTOR pathway inhibits ferroptosis through multiple effectors and warrants further investigation as a potential target for cancer treatment strategies involving ferroptosis induction

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Although the downstream response to c-MET is common to many RTKs, the potency, endurance and specificity of c-MET-triggered pathways is secured by a network of upstream signaling co-receptors that physically associate with c-MET at the cell surface (Figure 3) [Trusolino et al

(2019, July 9)