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The trend toward increased glutathione at Week 12 suggests sustained benefits of continued glycine supplementation

Molecular mechanisms of bone metastasis and associated muscle weakness

Decrease in glutathione (GSH) content in bovine sperm after cryopreservation: comparison between two extenders

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Zhou C, Zaman N, Li Y, Martinez-Arguelles DB, Papadopoulos V, Zirkin B, et al

It has been shown that upregulated MMPs in activated astrocytes in the ONH degrade the extracellular matrix (ECM) after retinal I/R injury in mice.[53] As a consequence the ECM is remodeled and degeneration of the lamina cribrosa occurs, which predisposes the RGCs towards compression[54, 55] and can cause further vascular dysregulation.[56] Indeed, we previously showed that the thickness of the lamina cribrosa, which indicates its degeneration, was related to BF in the ONH in glaucoma.[57] Astrocytes also release endothelin, which acts as a strong inducer of vasoconstriction[50] and prevents axonal transportation in the RGCs.[58] Furthermore, although NO in the astrocytes does not itself damage RGCs, it can be converted into the form of peroxynitrite (ONOO - )