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Once in the nucleus, it binds to specific transcription factors and DNA elements particularly antioxidant response elements (ARE) that are regulated by the stress-responsive factor NRF2. Through this mechanism, MOTS-c can turn on a suite of stress-defense and metabolic genes that help cells adapt to high-fat stress or insulin resistance

Its a deep, persistent exhaustion that doesn't go away with rest

Weeks 6-8: Peak Effect Window What's happening internally: Maximum fat oxidation response

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The other 80% goes to your small intestine and then into your bloodstream when your body then tries to get rid of it