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Negative charged threonine 95 of c-Jun is essential for c-Jun N-terminal kinase-dependent phosphorylation of threonine 91/93 and stress-induced c-Jun biological activity

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Additionally, while ALA has been thought to activate insulin signaling pathways to combat diabetes [28] and has been shown to prevent high fructose-induced cardiometabolic disorders and renal dysfunction [62], it has also been reported that ALA could only attenuate proteinuria and oxidative stress without slowing progression of diabetic renal failure [63]

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