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Acknowledgments We thank BioRender (www.biorender.com) providing mapping assistance during the preparation of this manuscript

Corrective Actions: If problems arise, the TGA may require recalls, safety notices, or fixes to reduce risk

We demonstrate that 1,6-HD effectively inhibits PSR-1 condensation in vivo and reduces axonal fusion triggered by overexpression of PSR-1

Automated syntheses were carried out on an Applied Biosystems 394 DNA/RNA synthesizer on 1-mol (CPG) and 5.25-mol (PS) scales

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Our findings revealed that exposure to OC upregulated the protein expression of AhpC, AhpF, and SOD in MRSE cells (Figure 7A), indicating that MRSE is attempting to enhance its enzymatic defense system in response to oxidative stress