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The main adducts, 8-OHdG and 8-oxo-7,8-dihydro-2-deoxyguanosine (8-oxodG) are formed when hydroxyl radicals are added to guanine

GHK-Cu stimulates the production of dermatan sulfate and glycosaminoglycans that hydrate and plump the dermal matrix, measurably improving skin thickness and density over treatment courses

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TABLE 4 Tissue and blood arsenic metabolites concentration following oral administration of ATO for 28 days

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Mechanistically, BPC-157 appears to function through nitric oxide-mediated vasodilation, angiogenic factor regulation, and recruitment of fibroblasts and endothelial progenitor cells 1-3