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According to the A-tau interaction hypothesis, A and tau pathology, which results in synaptic dysfunction, neuronal death, and cognitive impairment (96), is determined by the intrinsic interaction with A and tau (A42 enhances tau phosphorylation, truncation, and aggregation, while tau can increase A synthesis)

Supports investigation of tissue structural rejuvenation: Accumulated senescent cells can impair tissue architecture by altering cell-to-cell communication and extracellular matrix composition

One key challenge lies in bringing together these individually engineered components

Another study reported increased cortical and striatal GABA/glutamate ratios in a neurofibromatosis type 1 mouse model of autism 21

Some patients experience mild injection site reactions, transient changes in sleep patterns as circadian rhythms adjust, or minor digestive changes as metabolism optimizes

Myeloid deletion of nuclear factor erythroid 2-related factor 2 increases atherosclerosis and liver injury