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Proline metabolism has been confirmed to enhance the tumorigenesis in liver cancer as two enzymes corresponding to proline biosynthesis are upregulated (pyrroline-5-carboxylate reductase (PYCR1), aldehyde dehydrogenase 18 family member A1 (ALDH18A1)), and one proline catabolic enzyme is downregulated (proline dehydrogenase (PRODH)) ( Moreover, the interplays between metabolism and tumor microenvironment play crucial roles in the cancerous liver

Few studies have also developed an LS model for neuroprotection studies by using the complex I inhibitor MPTP (1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine)

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