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Published 2022 Feb 7

[0432] In certain embodiments, the anti-ALPP/ALPPL2 antibodies described herein {e.g., M25ADLF, M25ADLFEG, M25ADLFDS, M25FYIA, M25FYIAEG, M25FYIADS, M25, M25EG, M25DS, M25AELF, M25AELFEG, M25AELFDS, M25ADL99P, M25ADL99G, M25ADS95R, M25ADD28G, M25ADS91G, M25ADY93H, M25ADYHSRLF, M25GRITSGFYGDwtLC, M25FSITSGFYGDwtLC, M253018IA, M253018LF, M25AD, M25ADX, ALPPL2rd3_l, ALPPL2rd3_2, M25AGIA, M25AGLF, M25ASIA, M25ASLF, M25ASwt, M25AVIA, M25AVLF, M25ALIA, M25ALLF, M25wtIA, and/or M25wtLF) can be chemically conjugated to the effector molecule (e.g., a cytotoxin, a label, a ligand, a drug, a liposome, etc.)

Jung WK, Park SB, Yu HY, Kim YH, Kim J (2022b) Effect of esculetin on tert-butyl hydroperoxide-induced oxidative injury in retinal pigment epithelial cells in vitro

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Concurrently, repeated exposure to NIR may provoke cerebral oxidative stress, resulting in lipid peroxidation and DNA damage that synergistically amplifies AD-related oxidative pathology

(Table 2)