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Such [Ca 2+ ] i increase results in the activation of protein kinases and other downstream Ca 2+ -dependent enzymes that destroy important molecules and disintegrate the cell membrane, causing further Ca 2+ influx to the cells, release of free radicals from damaged mitochondria, and subsequent cell death (Chan, 2019

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Polycystic ovary syndrome and 25-hydroxyvitamin D: A bidirectional two-sample Mendelian randomization study

track down the events through which the probiotic Lactobacillus GG releases soluble molecules that in turn activate the p38 MAPK pathway that synthesizes heat-shock proteins to shield the intestinal epithelial cells from damage (Tao et al., 2006)

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