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The upregulated expression pattern of SLC7A11 suggests an active compensatory mechanism in response to oxidative stress, representing a potential intervention point where modulation could either enhance the protective response or redirect it toward more effective therapeutic outcomes

Clinical healing (reduced pain, increased function, weight-bearing tolerance) important markers

Moreover, EGCG therapy improved learning and memory in stressed rats and stopped the decline in locomotor activity [25]

R., Henry, A.-I., Dieringer, J

Yamamoto, M., Inohara, H

Gallic acid improved inflammation via NF-B pathway in TNBS-induced ulcerative colitis