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For example, the dependence of many cancer cells specifically on the G2/M checkpoint has led to the development of agents that target this checkpoint, particularly Chk1, Wee1, ATM, and ATR (Dillon et al., 2014)

sulfurreducens and with anaerobe bacteria in general (Fahey, 2013)

Sukhovskaya IV, Borvinskaya EV, Smirnov LP

10.3390/cells11081374 149 WolstenholmeJ

Exaggerated angiogenesis will increase but also restrain tumor access and become a pathological process per se [160]