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Therefore, it is thought that HERC2 deficiency would result in a malfunctioning response to elevated iron levels, resulting in ferritinophagy, free iron release, and neuronal cell damage

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As essential amino acids, elevated serum levels of branched-chain amino acids, including leucine, valine, and isoleucine, increase ROS production by activating the Akt-mTOR signaling pathway, thereby affecting ferroptosis [48]

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