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A selective ACAT-1 inhibitor, K-604, stimulates collagen production in cultured smooth muscle cells and alters plaque phenotype in apolipoprotein E-knockout mice

This shift has moved nanotherapeutic design from general exposure modification toward more explicit attempts to match carrier functions with phenotype-specific TME barriers

Although conventional or reduced-dose HMAs exhibit some activity in patients with LR-MDS, the limited activity and transient response of HMAs following the failure of ESAs as first-line therapy means they are seldom used [258]

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Antioxidant supplementation prevents exercise-induced lipid peroxidation, but not inflammation, in ultramarathon runners