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Description

Elevated HMGB1 levels have been detected in the blood of patients, where it interacts with receptors such as RAGE and TLR4, forming complexes with IL-1 or LPS to activate proinflammatory signaling pathways (Shimizu et al., 2017)

Multiple studies report that its effects on myoblast proliferation and migration are not abolished by blocking the IGF-1 receptor, implying that the isolated E-domain peptide acts through a different, non-IGF-1R route