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This protection mechanism may explain the poor performance of residual chlorine in controlling bacterial regrowth in DWDS 33 and the formation of biofilms 34 , especially considering the stimulation of the GSH by oligotrophic conditions and residual chlorine in DWDS (Table 2)

PMID: 39013742

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doi: 10.1093/toxsci/kfi336

To the best of our knowledge, this study is the first to identify miR-29b as a direct effector molecule mediating the antifibrotic effects of TSA, unveiling a novel pathway through which TCM active components target miRNAs to modulate cardiac remodeling