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1A, i.e., that the TOCC of the control sample (C) was significantly higher than that of the sample supplemented with sulfur dioxide (SO 2 )

found that in amyotrophic lateral sclerosis, the 43 kDa TAR DNA-binding protein, a marker protein, can lead to mPTP opening and mtDNA release into mitochondria

Limitations of the model This model was designed to allow us to study various regulatory mechanisms in the transsulfuration pathway and the effects of oxidative stress, particularly as applied to Down syndrome and autism

The ability of a quinone to redox cycle is dependent upon the favourability of its reduction by a single electron, which can be quantitatively described by its reduction potential ( E o )

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