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In addition, Irg1-deficient phagocytes, which cannot produce itaconate, have shown increased susceptibility to M.tb infection, with infected mice exhibiting higher bacterial loads, reduced survival, exacerbated inflammation, extensive necrotizing granuloma formation and elevated neutrophil infiltration in the lungs [104, 105]

Summary The prevailing view on the role of glutathione in protein disulphidebond formation is that this tripeptide acts as a net reductant in the ER, either by maintaining ER oxidoreductases in a reduced state or by directly reducing nonnative disulphide bonds in substrate folding proteins

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15 This association was not observed for females

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