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Simultaneous detection and quantification of mitochondrial DNA deletion(s), depletion, and over-replication in patients with mitochondrial disease

NK cells exhibit strong metabolic flexibility upon short-term cytokine stimulation, but prolonged activation leads to increased mitochondrial metabolism and glycolysis involving mammalian target of rapamycin complex 1 (mTORC1) ( Metabolomic and lipidomic studies have identified distinct alterations in glycerophospholipids among different NK cell subsets ( Figure 3 )

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Studies have revealed abnormal levels of lipids, amino acids, carbohydrates, and nucleotide metabolism in COPD patients, playing crucial roles in disease progression (Xu et al