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and socioeconomic factors, including occupational and recreational exposures

We found CHF could effectively suppressed clinical dose of trivalent arsenic of the kidney toxicity, and its molecular mechanism were associated with the inhibition of caspase three induced renal tubular epithelial cell apoptosis

37 NeitelerA.PalakkanA

Another experiment confirmed that H 2 S (as potentially formed by GSH-mediated GSSH reduction) is unlikely to be involved in radical scavenging: Na 2 S inhibited resorufin formation only weakly (Extended Data Fig

Maltotetraose, metabolite in the glucose metabolism pathway, were higher in pre-ART PLWH than in HCs and further increased after ART

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