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This interaction triggers the assembly of a signaling complex that includes interleukin-1 receptor-associated kinases and TNF receptor-associated factor 6, leading to the activation of nuclear factor kappa B (NF-B) and mitogen-activated protein kinases (MAPKs) such as p38 and JNK [128]

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Nitric oxide-dependent long-term potentiation is blocked by a specific inhibitor of soluble guanylyl cyclase

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Nada de inyectable sin datos

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[24] demonstrated high ASCT2 expression in the gastric tissues of patients with GC

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Antigen contact sites in class I major histocompatibility complex-restricted, trinitrophenyl-specific T cell receptors

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