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J.FedlerJ.CoffeyC

Parsanathan R, Jain SK

& Hegenbart, U

Consistent with reports showing that the topoisomerase II-inhibitor Etoposide is a substrate of ABCC1 42 , the GI 50 of MOLM-13 ABCC1 -knockout clones to Etoposide was 1.4 2.4-fold lower than in control cells (Supplementary Fig

GHK-Cus activation of DNA damage response networks (including ATM, BRCA1, and p53 pathways) in gene expression analyses positions it as a potential research subject for neurodegeneration prevention