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doi:10.1161/circulationaha.111.050773

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Roles of endoplasmic reticulum stress and autophagy on H2O2-induced oxidative stress injury in HepG2 cells

The discrepancy between the high frequency of remodeling events and the slow rate of ECM protein synthesis can be reconciled by the existence of a mobile, recycling pool of ECM molecules that can be continually incorporated and re-internalized at synapses, without the need for novel protein secretion, as we found here